Histometric analysis of gingival hyperplasia in Wistar rats during nifedipine administration

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منابع مشابه

Gingival hyperplasia with nifedipine.

36 Hind CR, Winearls CG, Lockwood CM, Rees AJ, Pepys MB. Objective monitoring of activity in Wegener's granulomatosis by protein concentration. Clin Nephrol 1984; 21:341-5. 37 Imbach P. Wegener'sche granulomatose. Ergeb Inn Med Kinderheilkd 1977;39:33-54. 38 Lehmann H, Neidermayer W. Stadienteilung der Wegener'schen granulomatose. Med Welt 1987;38: 466-9. 39 Lehmann H, Keifer B. Clinical manife...

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an investigation on gingival hyperplasia induced by nifedipine

statement of problem: in recent years, numerous reports have been presented in the literature, about gingival overgrowth induced by nifedipine- a calcium antagonist- prescribed for hypertension and certain types of angina. purpose: the purpose of the present study was to investigate the prevalence rate of nifedipine induced gingival hyperplasia in patients and its probable relationship with age...

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Therapy for nifedipine-induced gingival overgrowth by saireito in rats

OBJECTIVE A calcium antagonist, nifedipine, causes gingival overgrowth as a side effect. It has been confirmed that the Japanese traditional medicine, Saireito, inhibits the nifedipine-induced proliferation of gingival fibroblasts in vitro. We performed an in vivo experiment to determine whether Saireito has a therapeutic use in the treatment of nifedipine-induced gingival overgrowth. METHODS...

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Diltiazem did not induce gingival overgrowth in rats. A clinical, histological and histometric analysis.

The administration of calcium channel blockers has been associated with gingival overgrowth. However, there are few studies in humans or animals that evaluated the effect of diltiazem on gingival tissues. The present study assessed the influence of diltiazem, at different dosages and treatment duration, on gingival tissues of rats, using clinical, histological and histometric analyses. Eighty y...

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Nifedipine-induced gingival hypertrophy.

A 55-year-old man was referred to us because of renal dysfunction (serum creatinine 1.82 mg/dL). Three years earlier, he had aortic dissection associated with left renal artery occlusion and had undergone surgery for ascending aorta and aortic arch replacement. He had been receiving the following medications for 3 years; nifedipine (Adalat-CR) 40 mg, carvedilol 10 mg, aspirin 100 mg, allopurino...

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ژورنال

عنوان ژورنال: Military Medical and Pharmaceutical Journal of Serbia

سال: 2007

ISSN: 0042-8450,2406-0720

DOI: 10.2298/vsp0701019b